Immunogenetics of Endocrine DisordersNadir R. Farid |
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21-hydroxylase abortion activity addition adrenal alleles analysis antibodies antigens appears associated autoantibodies autoimmune thyroiditis blood cells chromosome class II Clin Endocrinol clinical common compared complex controls culture cytotoxicity deficiency detected determinants diabetes mellitus disease disorders effect endocrine et al evidence expression factors families Farid fragment frequency function gene genetic Genotype Graves haplotypes histocompatibility HLA-DR human hypothyroidism IDDM immune Immunol important increased indicated individuals induced insulin insulin receptors insulin-dependent diabetes interaction involved iron liability linkage loci locus lymphocytes major markers mice molecules mouse Nature normal observed occur patients phenotype polymorphism population possible pregnancy present probes rats recently receptor region relative reported response restriction risk role serum shared showed shown similar specific spontaneous stimulation strains studies suggested surface T-cell Table Tissue trophoblast