Lesion-Induced Neuronal Plasticity in Sensorimotor SystemsHans Flohr, W. Precht Sensorimotor systems are not rigidly wired predetermined networks but rather highly plastic structures that learn and modify their entire performance in response to changes in external or internal conditions. Lesions or distortions of the system's input, which initially cause a functional disorganization, induce an active reorganization which often leads to a recovery of function. Examples of lesion-induced neural plasticity have been known for some hundred years; however, an awareness of their value as research tools is relatively new. This current interest is a consequence of rapid ly changing ideas concerning the nature of CNS organization. Out of these, concepts are emerging which describe neural nets as modifiable, highly dynamic, self-organizing structures. This trend is clearly reflected in this volume, which contains the proceedings of a symposium held in Bremen in July 1980 as a satellite meeting of the XXVIIIth International Congress of Physiological Sciences. The first part of this conference was devoted to some gen eral aspects of plasticity, discussing the current theories of functional recovery as well as morphological, neurochemical, physiological, molecular, and ontogenetic aspects. The second part dealt with lesion induced plasticity in specific sensorimotor systems of the spinal cord, brain stem, and cerebral cortex. |
Contents
The Significance of Neuronal Cell Death During the Development | 3 |
R 75 | 10 |
Abeln W 153 265 | 38 |
Copyright | |
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Other editions - View all
Lesion-Induced Neuronal Plasticity in Sensorimotor Systems Hans Flohr,W. Precht No preview available - 2014 |
Lesion-Induced Neuronal Plasticity in Sensorimotor Systems H. Flohr,W. Precht No preview available - 2011 |
Common terms and phrases
abducens motoneurons abducens nucleus activity adult afferent animals asymmetry axonal sprouting axotomy Azzena Bechterew behavior bilateral central cerebellar cerebellum changes chronic collateral sprouting commissural conditioning lesion contralateral cortex Cotman CW deafferentation deafferented dendritic denervation dentate gyrus dorsal effects entorhinal enzyme EPSP EPSP amplitude EPSP's Exp Brain Res eye movements fibers flocculus frequency frog function gain head deviation head movements hemilabyrinthectomy hemisection hippocampus horizontal increase inferior olive innervation input intact side ipsilateral labyrinth labyrinthine locomotion McQuarrie mechanisms molecular layer motoneurons motor muscle nerve nervous system Neurophysiol Neurosci normal nystagmus observed optic optokinetic pathways phase Physiol plasticity postoperative posture Precht projection protein Purkinje cells rabbits recovery red nucleus regeneration response retinal rotation saccade Schaefer spared root spinal cord stimulation synaptic terminal testing lesion tion transection unilateral labyrinthectomy velocity vestibular compensation vestibular neurons vestibular nuclei vestibular system vestibulo-ocular reflex visual