Pathology of the Kidney, Volume 1 |
Contents
Congenital Malformations | 69 |
Hypertension I Essential | 121 |
Hypertension II Secondary | 163 |
Copyright | |
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activity acute acute glomerulonephritis affected antibody antigen appear areas associated basement membrane blood pressure capillary capillary walls cause cells changes chronic circulation Clin clinical complement condition considered course cytoplasm damage demonstrated deposits described develop diffuse disease early electron electron microscopy epithelial essential hypertension et al evidence experience experimental factors findings focal frequently function further glomerular glomeruli glomerulonephritis granular histologic human hypertension immune complex immunofluorescence important increased infarction infection Intern involved kidney later lesions less levels malignant material mechanism mesangial nephritis nephrosis nephrotic syndrome normal noted observations obstruction occur papillary necrosis Pathol pathologic patients pattern percent phase picture possible present produced progressive proliferative protein proteinuria rabbit renal artery renal failure renin reported responsible seen serum severe shown similar sometimes stage staining suggested thickening tissue tubular tubules tuft usually various vascular