The Pharmacological Basis of TherapeuticsDrugs acting on the central nervous system; Local anesthetics; Drugs acting at synaptic and neuroeffector junctional sites; Autacoids; Cardiovascular drugs; Water, salts, and ions; Drugs affecting renal function and electrolyte metabolism; Drugs affecting uterine motility; Gases and vapors; Heavy metals and heavy-metal antagonists; Locally acting drugs; Chemotherapy of parasitic diseases; Chemotherapy of microbial diseases; Chemotherapy of neoplastic diseases; Drugs acting on the blood and the blood-forming organs; Hormones and hormone antagonists; The vitamins. |
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Page 273
... antagonists such as nalorphine and cyclazocine produce their agonistic effects . Within the framework of a dual- or multi- ple - receptor hypothesis , a drug could have varying degrees of agonistic activity at the morphine recep- tor ...
... antagonists such as nalorphine and cyclazocine produce their agonistic effects . Within the framework of a dual- or multi- ple - receptor hypothesis , a drug could have varying degrees of agonistic activity at the morphine recep- tor ...
Page 276
... antagonists in reversing narcotic- induced respiratory depression in the adult have already been discussed . Narcotic antagonists have also been effectively employed to decrease neonatal respiratory depression secondary to the adminis ...
... antagonists in reversing narcotic- induced respiratory depression in the adult have already been discussed . Narcotic antagonists have also been effectively employed to decrease neonatal respiratory depression secondary to the adminis ...
Page 610
... antagonist is effective against the various other autacoids liberated or formed during allergic responses . Such substances are clearly more important than histamine in bron- chial asthma . In this disease , histamine antagonists are ...
... antagonist is effective against the various other autacoids liberated or formed during allergic responses . Such substances are clearly more important than histamine in bron- chial asthma . In this disease , histamine antagonists are ...
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absorption acid action activity acute addition administration agents alcohol amount analgesic anesthesia anesthetic animals appear associated barbiturates block blood body brain cardiac cause cells central changes Chapter chronic clinical compounds concentration containing convulsions decrease dependence depression develop direct discussed dosage doses drug effects employed enzyme epinephrine et al excreted factors fibers function given heart important increase individual inhibition initial injection intravenous involved less mechanism medication membrane metabolism morphine muscle narcotic nerve nervous normal observed occur opioid oral oxide pain patients period Pharmac pharmacological physical plasma poisoning possible potential preparations present pressure probably produce properties reactions receptors reduced relatively release reported respiratory response result salicylate seizures selective severe side similar solution spinal stimulation structure studies subjects symptoms Ther therapeutic therapy tion tissues tolerance toxic treatment usually various withdrawal