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cumstances the initial record of bladder function was made Figs 2 and 3 tracings
symptoms had started seven years before and became typical after four years and a half
ment with desiccated thyroid Figure 3 shows graphically the variations
17-ketosteroids abnormal acid ACTH activity administration adrenal cortex adrenocortical amounts amphenone androgen androsterone ascites Biol blood bone calcium cancer carcinoma cells cent Chem chromatography cirrhosis Clin clinical compounds concentration conjugated 17-OHCS corticosterone corticotropin Cortisol daily determined diagnosis disease dosage dose effect Endocrine Society Endocrinol Endocrinology eosinophil estrogen euthyroid exophthalmos extracts fraction goiter granulomatous thyroiditis histologic Hospital human hydrocortisone hypercalcemia hyperplasia hyperthyroidism hypoparathyroidism I131 uptake increased infusion injection intravenous iodide laboratory liver lobe magnesium measured Metab metabolism method mucoprotein myxedema normal subjects observed obtained parathyroid patients periodic paralysis phosphorus pituitary plasma Porter-Silber pregnanediol pregnanetriolone present protein protein-bound iodine radioactive iodine radioiodine reaction reported samples serum steroids symptoms syndrome Table testosterone tetrahydrocortisone therapy thyroglobulin thyroid gland thyroid hormone thyroidal I131 thyrotoxic thyrotropin thyroxine tion tissue treatment triiodothyronine tumor twenty-four hours uracil urine values