Inflammation: A Review of the Process
Quintessence Publishing Company, Jan 1, 1997 - Medical - 236 pages
Combining their backgrounds in pathology and education, Drs Trowbridge and Emling have created a comprehensive "self-study" program in inflammation. Chapters build on the reader's basic medical science background, and strategically placed "self-tests" allow readers to pause and test their comprehension before advancing to new material. Useful metaphors, informative illustrations, and a new clinical-connections section make this book an ideal refresher for busy clinicians and an indispensable study aid for graduate students preparing for specialty board examinations.
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Vascular Response to Injury
Chemical Mediators of the Vascular Response
The Blood Leukocytes
3 other sections not shown
abscess acute inflammation anti antibody antigen APCs arachidonic acid bacteria basophils bind blood vessels capillaries caries carious causes cell-mediated chemotactic factors chemotaxis chronic inflammation chronic inflammatory tissue clot collagen complement system component connective tissue CTLs cyst cytokines cytotoxic degranulation dentin edema endothelial cells enzymes eosinophils epithelial epithelium exudate fibers fibrin fibroblasts fibronectin fluid formation free radicals granuloma growth factor healing histamine hypersensitivity immune complexes immune response immune system immunoglobulin immunologic increased vascular permeability infection inflammation inflammatory cells inflammatory reaction inflammatory response Interleukin intracellular involved kinin lesion leukocytes leukotrienes lymphocytes lysosomal lysosomal enzymes macrophages mast cells mediators migrate molecules monocytes necrosis neutrophils NK cells occur odontoblasts organ pathway periapical granulomas periodontal disease phagocytic phagocytosis plasma cells platelets produce proliferation prostaglandins proteases proteins pulp pulpal receptors release repair role Self-test serum stimulate surface T-cell T-helper cells tion tooth tumor vascular response vasodilation venules wound