Evaluation of Abomasal Outflow Diversion as an Experimental Model of Hypochloremic, Hypokalemic Metabolic Alkalosis in Lactating Dairy Cows and Comparison of 0.9%, 3.6%, and 7.2% NaCl in Correcting Experimentally Induced Hypochloremic, Hypokalemic Metabolic Alkalosis in SheepCornell University, 1992 - 198 pages |
Contents
Hypertonic Saline for Correction | 13 |
Evaluation of Abomasal Outflow Diversion | 19 |
17 | 41 |
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Common terms and phrases
12 hours abomasal effluent abomasal outflow diversion abomasum acid acid-base base excess concentration bicarbonate cannula cattle Changes in plasma Circ Shock clinical CONCENTRATION mEq/L correction COW 1 COW decreased dehydration DIVERSION HOURS diversion of abomasal diversion period duodenum ECF volume electrolyte end of diversion excretion experimental model experimentally induced extracellular fluid fluids were administered following abomasal outflow following treatment Galla JH gastric gastrointestinal glomerular filtration rate hemorrhagic shock HHMA HHMA in sheep hour period hours of diversion hypertonic 3.6 hypertonic saline Hypochloremic hypokalemia hypokalemic metabolic alkalosis hyponatremia intravenous isotonic or hypertonic lactating Luke RG mEq/L metabolic alkalosis model of HHMA Muir WW NaCl Packed cell volume parameters period from END Physiol plasma chloride concentration plasma potassium concentration plasma sodium concentration POST-TREATMENT HOURS pre-diversion levels PRE=pre-diversion pylorus renal resulting from abomasal resuscitation sodium chloride surgical treatment fluids treatment with isotonic trials urine urine production Values represent mean volume expansion water intake