Goodman and Gilman's The Pharmacological Basis of Therapeutics |
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Page 846
... ( Chapter 65 ) ; iodide is presented with other agents that are relevant to the function of the thyroid gland ( Chapter 60 ) , and the salts of iron are discussed in the context of hematopoiesis ( Chapter 56 ) . Drugs that are inorganic ...
... ( Chapter 65 ) ; iodide is presented with other agents that are relevant to the function of the thyroid gland ( Chapter 60 ) , and the salts of iron are discussed in the context of hematopoiesis ( Chapter 56 ) . Drugs that are inorganic ...
Page 1675
... ( Chapter 50 ) a Nonlinear binding ; value decreases from 93 % at 25 μg / ml to 89 % at 250 μg / ml . b area is about 50 % larger than V ,, given here . CEFORANIDE ( Chapter 50 ) CL = 0.26 CLcr + 0.07 CEFOTAXIME ( Chapter 50 ) a Drug ...
... ( Chapter 50 ) a Nonlinear binding ; value decreases from 93 % at 25 μg / ml to 89 % at 250 μg / ml . b area is about 50 % larger than V ,, given here . CEFORANIDE ( Chapter 50 ) CL = 0.26 CLcr + 0.07 CEFOTAXIME ( Chapter 50 ) a Drug ...
Page 1708
... ( Chapter 51 ) SULFADIAZINE ( Chapter 49 ) a Study included concurrent administration of trimethoprim . SULFAMETHOXAZOLE ( Chapter 49 ) a Assuming 70 - kg weight . SULFISOXAZOLE ( Chapter 49 ) a Dependent on rate of urine formation and pH ...
... ( Chapter 51 ) SULFADIAZINE ( Chapter 49 ) a Study included concurrent administration of trimethoprim . SULFAMETHOXAZOLE ( Chapter 49 ) a Assuming 70 - kg weight . SULFISOXAZOLE ( Chapter 49 ) a Dependent on rate of urine formation and pH ...
Contents
SECTION | 1 |
Mechanisms of DRUG ACTION AND THE RELATION | 35 |
PRINCIPLES OF THERAPEUTICS | 49 |
Copyright | |
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absorption acid action activity acute administration adrenergic agonists alcohol amines analgesia analgesic anesthesia anesthetic antagonists anti-ChE agents antidepressants antipsychotic arterial atropine autonomic B-adrenergic barbiturates benzodiazepines binding blockade blocking agents blood flow blood pressure brain cardiac cardiovascular catecholamines cause cells central Chapter chlorpromazine cholinergic chronic Clin clinical compounds concentration creased decrease depression diazepam disease dopamine dosage doses enflurane enzyme epinephrine excreted fects fibers function ganglia ganglionic gastrointestinal halothane hepatic hypotension increase inhalation inhibition inhibitors injection interactions intravenous levodopa lithium local anesthetics mechanisms membrane metabolism metabolites morphine muscarinic nephrine nerve nervous system neuroleptic neuromuscular neurons nicotinic norepinephrine occur opioids oral oxygen patients pentazocine peripheral Pharmacol pharmacological phenobarbital phenytoin plasma potential produce propranolol protein receptors reduced release renal reserpine respiratory response result seizures side effects sleep smooth muscle spinal stimulation sympathetic sympathomimetic Symposium symptoms therapeutic therapy tion tissues tive toxicity transmitter treatment tricyclic tricyclic antidepressants usually