Muscle Relaxants |
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acetylcholine action activity administered agents altered amount Anaesth anesthesia Anesthesiology anesthetic animals anticholinesterase appears associated binding blood blood flow bound cardiac cause cell cent changes choline cleft clinical concentration contraction curare d-tubocurarine decamethonium demonstrated depolarizing depression disease distribution dogs dose drug duration effect end-plate et al evidence excretion experiments factors failure Feldman Figure gallamine given halothane heart increase injection ions isolated junction Katz less liver lower membrane minutes molecules motor nerve muscle relaxants neostigmine neuromuscular block neuromuscular transmission nondepolarizing relaxant normal observed occur onset pancuronium paralysis patients Pharmacol Phase Physiol plasma plasma level poison possible postsynaptic potassium potential present presynaptic produced prolonged protein rapid receptor recovery reduced relaxant drugs release renal reported resistance response result reversal seen sensitivity showed shown similar stimulation succinylcholine suggested synaptic technique tion twitch volume