Hypercalciuric States: Pathogenesis, Consequences, and TreatmentFredric L. Coe |
Contents
REABSORPTION OF CALCIUM BY | 13 |
INTESTINAL CALCIUM TRANSPORT | 37 |
RENAL PRODUCTION AND PERIPHERAL | 65 |
Copyright | |
9 other sections not shown
Common terms and phrases
abnormal acid acidosis acromegaly action activity administration appears associated balance bicarbonate Biochem bone resorption Calcif calcium absorption calcium excretion calcium transport cause cells changes chronic Clin Clin Invest clinical concentration decreased defect demonstrated diet dietary disease distal doses effect et al evidence factors failure finding fluid formation function glands growth human hypercalcemia hypercalciuria increased indicate intake intestinal jejunum kidney levels loss lower magnesium matrix measured mechanism membrane Metab metabolites mineral nephron normal observed occur osteoclasts osteoid parathyroid hormone patients percent phosphate phosphorus Physiol physiologic plasma possible present primary hyperparathyroidism production protein proximal rats reabsorption recently reduced regulation relatively renal renal tubular reported response rickets role sarcoidosis secretion segments serum calcium shown sodium stimulation stone studies subjects suggested syndrome Table therapy tissue treatment tubule urinary calcium urine values vitamin vitamin D vitro