The Vestibulo-ocular Reflex and VertigoJames A. Sharpe, Hugh O. Barber This volume reviews clinically relevant studies of the vestibulo-ocular reflex, the otolithic-ocular reflex, and related eye movements and highlights the practical applications of these findings in the management of patients with vertigo (dizziness and/or balance disturbance) and the design of exercise programmes for vestibular rehabillitation. The section on diagnosis and treatment of vertigo addresses conerns such as psychogenic dizziness, imbalance and falls in the aged, and perilymph fistula, and includes descriptions of outpatient and postoperative vestibular rehabilitation programmes and surgical treatment of benign postural vertigo. Nystagmus, a cardinal physical sign of vestibular disorder, is also discussed in detail. Other contributors describe new approaches to testing the vestibulo-ocular reflex and otolithic function. |
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Page 76
... lesion with peripheral labyrinthine and lateral medullary lesions ( Wallenberg's syn- drome ) . Because otolith - ocular projections from the vestibular nuclei cross the midline to ascend in the contralateral medial longi- tudinal ...
... lesion with peripheral labyrinthine and lateral medullary lesions ( Wallenberg's syn- drome ) . Because otolith - ocular projections from the vestibular nuclei cross the midline to ascend in the contralateral medial longi- tudinal ...
Page 134
... lesions . It was pro- duced experimentally by Magendie in 1826 and Hertwig in 1824 after surgical lesions apparently to the middle cerebellar pedun- cle and attributed to lesions there in pa- tients reported by Smith et al . ( 41 ) ...
... lesions . It was pro- duced experimentally by Magendie in 1826 and Hertwig in 1824 after surgical lesions apparently to the middle cerebellar pedun- cle and attributed to lesions there in pa- tients reported by Smith et al . ( 41 ) ...
Page 226
... lesions were due to nerve sections , they had no possibility of recover- ing function on the side of the lesion . It is currently thought that HSN2 is due to cen- tral or peripheral vestibular adaptation . There are several situations ...
... lesions were due to nerve sections , they had no possibility of recover- ing function on the side of the lesion . It is currently thought that HSN2 is due to cen- tral or peripheral vestibular adaptation . There are several situations ...
Contents
I | 3 |
Clinical Anatomic and Physiologic | 15 |
Clinical Tests of the VestibuloOcular Reflex | 41 |
Copyright | |
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Common terms and phrases
Acta Otolaryngol afferents Ann Neurol artery asymmetry axis Baloh RW bilateral brainstem cause cells central cerebellar cerebellum clinical component contralateral deficits degrees/sec direction disease disorders dizziness Exp Brain Res eye move eye movements eye position eye velocity fixation flocculus fovea frequency function gain gaze head movement head rotation head velocity head-shaking HOKAN Honrubia horizontal human hypertropia input ipsilateral labyrinth latency lesions LMLN mechanism medial Meniere's disease ments monkey motor msec nerve Neurology Neurophysiol normal subjects nystag oculomotor OKAN Ophthalmol optokinetic nystagmus otolith organs pathway patients peripheral vestibular posterior postural rectus response roll-tilt saccades semicircular canal sensory signals sinusoidal skew deviation slow phase smooth eye smooth pursuit sponse stimulation Stockh strabismus symptoms syndrome tagmus target tibular tients tion torsion velocity storage vertical vertigo vestib vestibular neurons vestibular nucleus vestibular system vestibulo-ocular reflex Vision Res visual waveform Zee DS