Acute catabolic state
This book details the pathophysiology of the acute catabolic state and the therapeutic options available. It focuses in particular on the changes that take place in the early stage of the catabolic state, within and between different organ systems. International experts describe metabolic changes that are of the utmost importance for the outcome in patients with acute illness or trauma. There is also detailed discussion of possible strategies for modifying the acute response, the use of which has substantially changed available treatment in surgery and other catablic states. Possible future investigational strategies are outlined and discussed.
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Substrate Handling in StressInduced Catabolic State
The Early Endocrine Response to Injury
21201 9038 Troms0
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ACTH activity acute catabolic acute phase acute renal failure adrenal aldosterone amino acid Ann Surg antioxidants blood bone calcium catabolic catecholamines circulating Clin Invest clinical concentrations cortisol critically ill cyclooxygenase cytokines decrease effects endocrine endogenous endothelial cells endothelium endotoxemia endotoxin epidural epinephrine free radicals function glucagon gluconeogenesis glucose glutamine growth factors growth hormone hemorrhage hepatic human hypoglycemia hypothalamus increased induced infection inflammatory infusion inhibition inhibitor insulin interleukin-1 intestinal kidney levels lipid liver lungs macrophages mechanisms mediators membrane metabolic metabolic response modulation muscle protein nervous system neural neuroendocrine nitrogen balance normal Nutr opioid oxidation oxygen parenteral nutrition patients peptides Physiol physiologic pituitary plasma postoperative production prostaglandins protein synthesis pulmonary receptors regulation release response to injury rhGH role secretion sepsis septic shock skeletal muscle somatostatin stimulates stress studies substrate surgery surgical therapy tion tissue TNF-a trauma tumor necrosis factor uptake vascular Wilmore DW