Endocrinology of HypertensionF. Mantero, Edward G. Biglieri, Christopher Richard Watkin Edwards |
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by J A Clements J W Funder B A K Khalid Z Krozowski | 1 |
Peptidergic and monoaminergic regulation of aldosterone secretion | 11 |
New regulators of aldosterone secretion | 19 |
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100 PLASMA RENIN 200 BLOOD PRESSURE 50 CUMULATIVE SODIUM according to vasoconstriction-volume achieve normotension ACTH ACTIVITY 40 ng/ml/hr adding a renin ade produced dramatic aldosterone ANGIOTENSIN I INHIBITOR angiotensin II balance ward conditions BLOOD PRESSURE 150 captopril cortisol defended from volume diuretic therapy Endocrinology enzyme inhibitor teprotide essential hypertension essential hypertension differs factor Human factor Human essential forms of essential forms of renovascular furosemide glucocorticoid hemodilution Laragh high-renin forms high-renin patients hyperaldosteronism Hypertension The volume indomethacin INHIBITOR µg/kg/min kallikrein low-renin patients tend ng/ml/h partially to volume patients with essential plasma renin activity plasma renin levels produced partial correction promptly by adding prostaglandins reaction to elevated reactive plasma renin renin forms renin system renin system blocker renovascular hypertension respond to diuretic saralasin saralasin infusion Saralasin produced partial situations sodium depletion sodium chloride sodium-volume support strating first vasoconstriction subgroup responds especially superimposing a furosemide-induced vasoconstriction Vaughan et al volume depletion volume status