Metabolic Aspects of Chronic Liver Disease
Ami Schattner, Hilla Knobler
Nova Biomedical Books, 2008 - Liver - 362 pages
This book discusses in detail new aspects of the metabolic basis of important chronic liver diseases. Entities such as non-alcoholic fatty liver disease (fatty liver and Non-Alcoholic SteatoHepatitis -- NASH), diabeties in chronic hepetitis C, hemochromatosis, Wilson's disease, Gaucher disease, porphyria, as well as liver cirrhosis and its metabolic consequences will be discussed in detail. These clinical conditions are highly prevalent and affect millions of patients in the USA and world-wide. For example, non-alcoholic fatty liver disease is the most common cause of elevated liver enzymes in the general population. This field has been practically transformed over the last few years, with many new insights gained, regarding both pathogenesis and effective novel treatments.
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Pathophysiology of NASH
The Treatment of NonAIcoholic Fatty Liver
The Hepatitis C Virus and Diabetes Mellitus
8 other sections not shown
abnormalities accumulation activity adiponectin adipose tissue ai-antitrypsin deficiency aminotransferase antitrypsin associated biochemical Biol causes cells Chem chronic hepatitis chronic liver disease cirrhosis Clin clinical copper cytokines decreased diabetes mellitus diagnosis disorder dysfunction effects elevated enzyme fatty acids fatty liver disease ferritin free fatty acids function Gastroenterol Gastroenterology Gaucher disease gene genetic glucose glycogen storage disease hepatic iron hepatic steatosis hepatitis C virus hepatocellular carcinoma hepatocytes Hepatol Hepatology hepcidin hereditary hemochromatosis histological human hypoglycemia improvement increased induced inflammation inhibition insulin resistance insulin sensitivity interferon iron overload kinase leptin levels lipid peroxidation liver biopsy liver transplantation Metab metabolic syndrome mice mitochondrial muscle neurologic nonalcoholic fatty liver nonalcoholic steatohepatitis normal obesity oxidative stress pathogenesis patients with chronic patients with NAFLD patients with NASH Pediatr penicillamine phenotype phosphorylase plasma prevalence protein receptor reported role serum synthesis therapy TNF-a transferrin treatment triglyceride Wilson disease