Heart hypertrophy and failure
Heart Hypertrophy and Failure brings together leading basic scientists and clinicians, presenting improved knowledge of the pathophysiology and treatment of the condition. The result is a synthesis of state-of-the-art information on molecular biology, cellular physiology and structure-function relationships in the cardiovascular system in health and disease. The papers presented describe fundamental mechanisms underlying changes in the cellular machinery during the development of cardiac hypertrophy and heart failure. Audience: Students, scientists, clinical and experimental cardiologists who seek to understand and manage the perplexing problems of hypertrophy and heart failure.
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A GENETIC AND MOLECULAR EVENTS
Intracellular signaling and genetic reprogramming during development
Phenotypic changes in experimental cardiac hypertrophy and failure in relation
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Acad Sci USA activity adenylyl cyclase adrenergic agonists alterations angiotensin ATPase atrial Biochem Biol Chem Ca2+ uptake Ca2+-ATPase Caj transient calcium cardiac fibroblasts cardiac hypertrophy cardiac myocytes cardiomyocytes cDNA cellular Circ Res clones collagen collagenase congestive heart failure contractile creatine kinase decreased deletion diabetic diastolic dilated cardiomyopathy disease dystrophin effects enalapril enzyme extracellular failing hearts failing human heart familial hypertrophic fetal Figure function G-protein gene expression genetic growth factor hamster heavy chain human myocardium hypertensive hypertrophic cardiomyopathy increased induced inhibition isoform isoproterenol left ventricle left ventricular levels LVFW mechanisms membrane molecular mRNA mtDNA muscular dystrophy mutations myocardial infarction myocardium myosin Natl Acad Sci neonatal nonfailing norepinephrine nucleotide P-adrenergic P2AR stimulation pathway patients perfused phenotype phospholamban phosphorylation Physiol PMHC Proc Natl Acad protein kinase rat heart receptor regulation response role sarcoplasmic reticulum sequence SERCA2 studies synthesis TGF-Pi tissue transcription ventricular myocytes vitro ZINT