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AcidBase Balance during Alkalosis 20
l0 Urea Clearance Shortly after Alkalosis
Serum Electrolytes during Acute Alkalosis Induced
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acid acid-base balance acute administration albumin alka alkali kidney alkali therapy aluminum hydroxide aluminum phosphate arteries average normal azotemia blood pressure blood urea nitrogen calcium carbonate calcium carbonate therapy cells changes chloride content chloride loss clinical collecting tubules control kidney wt cortex daily decreased dehydration diet distilled water duodenal ulcer edema electrolytes epithelium feces fluid gastric aspiration gastric contents gastric juice gastric secretion glomeruli granular hospital day hypertension hypochloremia increased ingestion intake intravenously kidney losis loss of chloride marked markedly massive hemorrhage measured medulla metabolism microscopic mild mineral excretion mM/L moderate Necropsy Necropsy revealed normal saline noted observed occasional output parenteral peptic ulcer period phosphorus Physiol plasma potassium protein remained normal renal function retention rH rH serum chloride serum COg serum electrolytes severe alkalosis slightly sodium bicarbonate sodium chloride stomach tion total base urea clearance urinary urine values varied vomiting