Autism, Brain, and Environment
The increasing number of people being diagnosed with autism spectrum disorders (ASDs) cannot simply be explained by changes in diagnostic criteria or greater awareness of the condition. In this controversial new book, Richard Lathe contends that the recent rise in cases of ASDs is a result of increased exposure to environmental toxicity combined with genetic predisposition. Autism, Brain, and Environment proposes that autism is a disorder of the limbic brain, which is damaged by toxic heavy metals present in the environment. Lathe argues that most ASD children have additional physiological problems and that these, far from being separate from the psychiatric aspects of ASD, can produce and exacerbate the condition. This important and groundbreaking text provides a closely-argued scientific case for the involvement of both environmental and physiological factors in autism. Lathe's argument will also have a direct impact on treatment strategies and options. It will be of great interest to the scientific community, professionals, researchers, political and environmental lobbyists, teachers, psychologists, and parents and people with ASDs.
What people are saying - Write a review
We haven't found any reviews in the usual places.
Focus on the Limbic System
Chapter 6 Limbic Dysfunction Correlates with the Autistic Phenotype
Chapter 7 Environmental Factors Heavy Metals and Brain Function
Physiological Dysregulation in Autism
abnormalities acid activity Adolesc adult amygdala androgen anxiety ASD children ASD subjects Asperger associated Autism Dev autism spectrum disorders autistic children autistic disorder behavioral biochemical Biol blood brain regions cells Chapter Child Neurol childhood children with autism chronic Clin contribute cortex cortisol cytokine deficits dentate gyrus detoxification diagnostic dietary disease effects elevated environmental enzyme epilepsy etal evidence excess Figure function gastrointestinal gene genetic Gillberg glucocorticoid heavy metal heavy metal toxicity heme hippocampus homocysteine hormone human hyperactivity immune impaired increase infantile autism infection inflammation interleukin-1 lesions levels limbic brain limbic damage markers melatonin memory mercury metabolism methionine methylmercury mice molecule neuronal Neurosci oxytocin pathways patients Pediatr pervasive developmental disorders Pharmacol phenotype physiological porphyrins prevalence protein Psychiatry Psychol rats receptor reported risk factor role seen in ASD seizure serotonin social specific steroid stress sulfate syndrome temporal lobe therapy tion Toxicol trimethyltin tryptophan twins urinary