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Structure of the Liver
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abnormalities acid active active hepatitis acute agent alcoholic alterations antibody antigen appear ascites associated bile bile acids bilirubin Biochem biopsy bleeding blood cause cells changes cholestasis chronic chronic active Clin clinical collagen common concentration conjugated contain cytoplasm decreased detected diagnosis drug duct early effect elevated encephalopathy enzyme ethanol evidence excretion factors failure fatty fibrosis Figure findings flow formation function Gastroenterology glucose HBsAg hepatocellular hepatocytes human increased indicate infection injury Intern involved jaundice lesion levels liver disease measured mechanism membrane metabolism necrosis non-A non-B normal observed occurs Pathol patients plasma portal present primary biliary cirrhosis produced Progress protein reaction reduced renal reported response role seen serum severe similar specific studies suggest surface syndrome synthesis tests therapy tion tissue tracts treatment usually varices vein virus York