Intensive Care and Clinical Biochemistry
ACB Venture, Jan 1, 1994 - Science - 162 pages
An approach to understanding the metabolic derangements found in the critically ill, this book attempts to place the role of clinical biochemistry in context. It begins with a brief description of the cellular consequences of shock defined as an interruption in the delivery of nutrients to and metabolites from the cell. From there, the effects of shock are extended to individual organs and finally to the patient as a whole in the form of case studies.
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Delivery and utilisation of cellular nutrients in shock
0rgans in shock 62
4 other sections not shown
acetyl coenzyme acidosis adequate adrenaline albumin alveolar amino acids angiotensin aspartate transaminase assessment bilirubin biochemical blood pressure brain C-reactive protein capillary carbon dioxide cardiac output cardiovascular catecholamines cause cells cellular central venous circulation clinical coenzyme coma concentrations cortisol creatinine critically ill patients decreased dysfunction energy excretion factors fatty acids fluid balance fulminant hepatic failure glucagon gluconeogenesis glucose glycogen haemoglobin hormone hydrogen ions hypotension hypovolaemia hypoxia increased inotropes insulin interstitial intestinal intracellular IU/L kidney laboratory lactate liver lungs measurements membrane metabolic mmHg mmol/L monitoring multiple organ failure NAD+ nitrogen normal nutrients nutritional support oedema osmolality oxygen content oxygen delivery parenteral perfusion phosphate plasma urea potassium protein proteolysis pulmonary artery pulse oximeter pyruvate reabsorption reduced release renal failure respiratory response saturation sepsis shock shunt skeletal muscle sodium substrates synthesis Table tion tissues tubular urea urinary vascular vasoconstriction vasodilatation venous pressure ventilation