Methods in Biliary Research
This exciting new book brings together the most significant information on the procedures that have generated most of the recent progress in hepatobiliary pathophysiology. The authors describe their unique experiences, including specific technical and analytical problems. Since workers in the field often specialize within a limited area, the methodology covering each of the main aspects in hepatobiliary research is particularly useful. The book also includes an exhaustive reference list.
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Pathophysiology of Bile Secretion Mechanisms of Cholestasis
Biochemical Methods to Study Hepatic Cholesterol Metabolism
Methods for the Measurement of Cholesterol and Bile Acid Kinetics in Humans
Analysis of Phospholipids
Acta aggregation analysis aqueous assay Berk bile acid synthesis bile acids bile salt bile samples Bile Secretion biliary lipid Biochim Biol Biophys BS-L canalicular Carey carriers CDCA cells Ch crystals Chem chenodeoxycholic acid cholestasis cholesterol gallstones cholesterol synthesis cholic acid chromatography Clin column concentration conjugated detection determination dissolution enrichment enterohepatic circulation enzymatic enzyme equilibrium esters formation fraction gallbladder gallbladder bile gallstone Gastroenterology hepatic Hepatic Transport hepatocyte Hepatology HMG-CoA reductase HMG-CoA reductase activity HPLC human bile intracellular isotope kinetics light scattering Lipid Res liver measurement membrane method mevalonate micellar micelles microscopy microsomal mixed micelles model bile molecular species molecules monohydrate monomers native bile nucleation pathway patients phase phosphatidylcholine phospholipids plasma pool procedure proteins quantitative rat liver ratio Raven Press separation serum sinusoidal solubility solvent sterol studies substrate supersaturated synthesis rates taurocholate Tavoloni technique Transport and Bile ultracentrifugation uptake ursodeoxycholic acid vesicles vesicular vivo
Page 30 - Homozygous disruption of the murine mdr2 P-glycoprotein gene leads to a complete absence of phospholipid from bile and to liver disease. Cell, 75: 451-462, 1993.