Molecular Mechanisms of Cardiac Hypertrophy and Failure
This title reviews current knowledge of the mechanisms contributing to heart failure. Editor Richard Walsh and an internationally renowned team of contributors discuss key advances in molecular and cell biology, biochemistry, and pharmacology, focusing on advances that have a direct bearing on current clinical studies. It highlights developments ac
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abnormalities Acad Sci USA acid actin activation adrenergic agonist altered angiotensin apoptosis arrhythmia associated atrial binding Biol Chem Ca2+ channel calcium cardiac hypertrophy cardiac myocytes cardiomyocytes Cardiovasc Res cardiovascular caspase cause cellular chronic Circ Circ Res Circulation Clin Invest clinical Coll Cardiol collagen complex contractile coronary defects dilated cardiomyopathy disease dysfunction dystrophin effects encoding familial hypertrophic filament function gap junction gene expression genetic Heart Circ Physiol heart failure hypertension hypertrophic cardiomyopathy increased induced inhibition interactions intracellular ischemic isoform left ventricular left ventricular hypertrophy mechanisms mediated membrane mitochondrial molecular mouse muscular dystrophy mutations myocardial infarction myocardium myofilament myosin Natl Acad Sci overexpression patients phenotype phospholamban phosphorylation polymorphism Proc Natl Acad protein kinase receptor regulation remodeling response role ryanodine receptor sarcoglycan sarcomere SCN5A signaling pathways skeletal muscle structure studies subunit syndrome systolic tissue transcription factors transgenic transgenic mice troponin ventricle ventricular myocytes