The End of Alzheimer's: The First Program to Prevent and Reverse Cognitive DeclineThe instant New York Times and Wall Street Journal bestseller A groundbreaking plan to prevent and reverse Alzheimer’s Disease that fundamentally changes how we understand cognitive decline. Everyone knows someone who has survived cancer, but until now no one knows anyone who has survived Alzheimer's Disease. In this paradigm shifting book, Dale Bredesen, MD, offers real hope to anyone looking to prevent and even reverse Alzheimer's Disease and cognitive decline. Revealing that AD is not one condition, as it is currently treated, but three, The End of Alzheimer’s outlines 36 metabolic factors (micronutrients, hormone levels, sleep) that can trigger "downsizing" in the brain. The protocol shows us how to rebalance these factors using lifestyle modifications like taking B12, eliminating gluten, or improving oral hygiene. The results are impressive. Of the first ten patients on the protocol, nine displayed significant improvement with 3-6 months; since then the protocol has yielded similar results with hundreds more. Now, The End of Alzheimer’s brings new hope to a broad audience of patients, caregivers, physicians, and treatment centers with a fascinating look inside the science and a complete step-by-step plan that fundamentally changes how we treat and even think about AD. |
Contents
Disrupting Dementia | 3 |
Patient Zero | 21 |
How Does It Feel to Come Back from Dementia? | 31 |
A Primer | 41 |
From Bedside to Bench and Back | 55 |
The God Gene and the Three Types of Alzheimers Disease | 93 |
PART THREE | 113 |
Other editions - View all
The End of Alzheimer’s: The First Programme to Prevent and Reverse the ... Dr Dale Bredesen No preview available - 2017 |
The End of Alzheimer's: The First Program to Prevent and Reverse Cognitive ... Dale Bredesen No preview available - 2017 |
Common terms and phrases
23andMe acid active Alzhei Alzheimer's disease amyloid plaques amyloid-beta ApoE ApoE4 B₁₂ bacteria blood blood-brain barrier brain Bredesen called cancer cause cells chronic contributors copper cortisol Cyrex Array damage dementia dependence receptor developed diet drug effects estradiol evaluation exposure factors fats foods function genes genetic glucose gluten heimer's homocysteine hormone hs-CRP immune system improvement increase infections inflammation inflammatory insulin resistance ketosis leaky gut levels Lyme disease memory loss mer's disease mercury metabolic microbiome mold molecules mycotoxins neurodegenerative neurons neurotrophin normal optimal patients percent percentile PET scan physician plasticity balance pregnenolone prevent probiotics problem progesterone protein ratio ReCODE protocol reduce response reverse cognitive decline risk sensitivity signaling sleep apnea stress subjective cognitive impairment suboptimal subtype sugar symptoms synapses synaptoclastic testosterone tests thyroid tion toxic toxins treatment trophic tropisetron typically vitamin vitamin D zinc