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THE BLOODCLOTTING MECHANISM
PREGNANCY AND PLUMPNESS
ART IN BEDLAM
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abnormal acatalasia acetyl activates Factor activation of prothrombin additive or residue amount animals apnoea average become Bethlem Hospital bimodal biochemical blood group body water body weight calcium catalase check-ups chromosome cinchocaine classical theory clinical clotting factors clotting mechanism coagulation components concentration curve Dadd derivative theory diabetes dibucaine disease dose effect enzyme extra fact Factor X Factors VII Family studies fibrin fibrinogen food additives free drug gain during pregnancy genetic origin haemoglobin haemoglobin Zurich haemolytic haemophilia human hypocatalasic increase individuals inherited inhibited insulin intake isoniazid MATERNAL STORES metabolism molecules normal observed occurs oedema oedema fluid parity patients pattern phenotypes plasma polymorphism population pre-eclampsia pregnancy present pro-enzyme probably produce prothrombin activator pseudocholinesterase rapid inactivators responses revealed Richard Dadd seems sequence sequential theory showed shown in Fig slow inactivators storage substances sulphadimidine sulphonamide surface contact suxamethonium thrombin thromboplastin tissue thromboplastin toxic trypsin tyramine weeks of pregnancy weight gain weight-for-height women