Nutrition in the Prevention and Treatment of Abdominal Obesity

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Ronald Ross Watson
Elsevier, Feb 26, 2014 - Medical - 560 pages

Nutrition in the Prevention and Treatment of Abdominal Obesity focuses on the important roles that exercise, dietary changes, and foods play in promoting as well as reducing visceral fat. Nutritionists, dieticians, and healthcare providers seeking to address the abdominal obesity epidemic will use this comprehensive resource as a tool in their long-term goal of preventing chronic diseases, especially heart, vascular, and diabetic diseases.

Experts from a broad range of disciplines are involved in dealing with the consequences of excessive abdominal fat: cardiology, diabetes research, studies of lipids, endocrinology and metabolism, nutrition, obesity, and exercise physiology. They have contributed chapters that define a range of dietary approaches to reducing risk and associated chronic diseases. They begin by defining visceral obesity and its major outcomes; they also discuss the importance and the challenges of dietary approaches to reduce abdominal obesity, as compared to clinical approaches, with major costs and risks.

  • Offers detailed, well-documented reviews outlining the various dietary approaches to visceral obesity with their benefits and failures
  • Includes chapters on types of foods, exercise, and supplements in reducing obesity and its chronic clinical companions, especially diabetes and cardiovascular disease
  • Helps nutritionists, dieticians, and healthcare providers approach patients in making decision about nutritional therapies and clinical treatments for abdominal obesity, from an evidence-based perspective
 

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Authoritative and extensively footnoted, this book is a very good and extensive review of current and past literature. It somewhat fails in its critique of poorly-conducted studies that have consistently crept into so called-nutrition science. The tenor of the book seems to be to judge most results through a singular lens of the gluttony and slothfulness view of obesity, which is presented several times as a common sense given. There is strikingly limited content on the massive literature supporting the understanding of insulin as a major contributor to deposit and storage of fat in adipocytes and the corresponding blockage of release of fats from these tissues. A somewhat antiquated view of low carbohydrate diets is presented, with modest attention paid to the burgeoning literature on this subject. The role of fructose and its metabolic influences, for example, are not prominently in evidence. In attempting to achieve balanced coverage, the authors include useful reviews and extensive citations that make this a welcome reference work on the title subject and a springboard for a researcher seeking to review the original sources for content. A suggestion to such audiences is to seek out these original works to determine the degree of credence to be attributed to their authors' conclusions based on a critical evaluation of the scientific method and experimental design employed. 

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About the author (2014)

Ronald Ross Watson, PhD, is Professor of Health Promotion Sciences at the University of Arizona, Mel and Enid Zuckerman College of Public Health. Dr. Watson began his research in public health at the Harvard School of Public Health as a Fellow in 1971 doing field work on vaccines in Saudi Arabia. He has done clinical studies in Colombia, Iran, Egypt, Saudi Arabia and the United States which provides a broad international view of public health. He has served in the military reserve hospital for 17 years with extensive training in medical responses to disasters as the chief biochemistry officer of a general hospital, retiring as a Lt. Colonel. He is a distinguished member of several national and international nutrition, immunology, and cancer societies. Dr. Watson’s career has involved studying many lifestyle aspects for their uses in health promotion. He has edited over 100 biomedical reference books and 450 papers and chapters. His teaching and research focuses on alcohol, tobacco, and drugs of abuse in heart function and disease in mouse models.

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