Cellular interactions in cardiac pathophysiology
Despite the considerable success in treating diseases of the heart and blood vessels, they still remain the major cause of mortality throughout the world. One of the reasons underlying this problem is our lack of understanding of the molecular and cellular aspects of the processes involved. These problems are fully discussed in Cellular Interactions in Cardiac Pathophysiology, which draws together 25 contributions from leading investigators from all parts of the world. The contributions are grouped under three headings: Extracellular matrix and cardiocyte interaction; Myocytic adaptations and myocardial injury; and Signal transduction.
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An International Journal for Chemical Biology in Health and Disease
Cellcell and cellmatrix adhesion molecules in human heart and lung transplants 2127
Chronic hibernating myocardium
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ACE inhibitors adenylyl cyclase ADP-ribosylation adrenergic adrenoceptor angiogenesis angiotensin animals antibody antioxidant aortic arrhythmias ATPase ATPase activity atrial blood pressure canine capillary growth cardiac hypertrophy cardiomyocytes cardiomyopathy Cardiovasc Res cardiovascular catecholamines cellular changes chronic hibernating Circ Res Circulation collagen concentration contractile coronary artery cytokines decrease Dhalla dogs dose effect enalapril endogenous endothelial cells enzyme extracellular matrix fibroblasts fibronectin fraction function G-proteins growth factor heart failure heart rate human hypertension hypoxia increase incubated induced infarction infusion inhibition inotropic interstitial fibrosis ischemia ischemic isolated isoproterenol left ventricle left ventricular leukocyte mechanisms membrane metabolism metoprolol Mol Cell Biochem Mol Cell Cardiol Molecular mRNA muscle myocardial myocardial infarction myocardium myocytes Na/K)-ATPase neutrophils occlusion oxidative P-adrenoceptors patients Pelouch perfusion pertussis toxin Pharmacol Physiol preconditioning rabbit ramipril rat heart receptor reperfusion role sarcolemma significantly staining stimulation stress tion tissue transplant treatment vascular xanthine oxidase