The Biology of Human Longevity: Inflammation, Nutrition, and Aging in the Evolution of Lifespans
Written by Caleb Finch, one of the leading scientists of our time, The Biology of Human Longevity: Inflammation, Nutrition, and Aging in the Evolution of Lifespans synthesizes several decades of top research on the topic of human aging and longevity particularly on the recent theories of inflammation and its effects on human health. The book expands a number of existing major theories, including the Barker theory of fetal origins of adult disease to consider the role of inflammation and Harmon's free radical theory of aging to include inflammatory damage. Future increases in lifespan are challenged by the obesity epidemic and spreading global infections which may reverse the gains made in lowering inflammatory exposure. This timely and topical book will be of interest to anyone studying aging from any scientific angle.
What people are saying - Write a review
We haven't found any reviews in the usual places.
Other editions - View all
Acad Sci USA accelerated acid activity acute phase adult aging alleles Alzheimer disease Alzheimer’s disease amyloid apoE apoE4 apolipoprotein arterial associated atheromas atherosclerosis attenuated Biol birth weight blood brain bystander damage C-reactive protein Caenorhabditis elegans caloric caloric restriction cancer cause cells Chapter cholesterol chronic Clin cognitive coronary cytokines decreased dementia diabetes diet restriction dietary dwarf effects elevated endothelial energy estrogen evolution famine fetal growth Finch gene expression genetic Gerontol glucose hormone human immune impaired infections inflammation inflammatory influence insulin insulin-like intake interactions leptin lesions levels lipid longevity lower macrophages maternal metabolic mice models mortality mouse mutants Natl Acad Sci neurons normal nutrition obesity oxidative damage oxidative stress pathogens pathways peptide plaques plasma populations pregnancy Proc Natl Acad protein rats receptor regulation reproductive responses risk factors rodents role Section senescence studies telomere tissue transgenic vascular disease worm