Casarett & Doull's Toxicology: The Basic Science of Poisons, Seventh Edition
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Page 108
... Risk assessment/risk management framework. This framework shows in blue the four key steps of risk assessment: hazard identification, dose–response assessment, exposure assessment, and risk characterization. It shows an interactive, two ...
... Risk assessment/risk management framework. This framework shows in blue the four key steps of risk assessment: hazard identification, dose–response assessment, exposure assessment, and risk characterization. It shows an interactive, two ...
Page 109
... Hazard Evaluation of Pesticides type. Table 4.1 Objectives of Risk Assessment 1. Balance risks and benefits. Drugs Pesticides 2. Set target levels of risk. Food contaminants Water pollutants 3. Set priorities for program activities ...
... Hazard Evaluation of Pesticides type. Table 4.1 Objectives of Risk Assessment 1. Balance risks and benefits. Drugs Pesticides 2. Set target levels of risk. Food contaminants Water pollutants 3. Set priorities for program activities ...
Page 121
... risk. B. Probabilistic exposure methods for calculating the incremental lifetime cancer risk (ILCR) from arsenic ingestion. In this example, the soil concentration ... risk assessment issues. The WHO has. CHAPTER 4 RISK ASSESSMENT 121.
... risk. B. Probabilistic exposure methods for calculating the incremental lifetime cancer risk (ILCR) from arsenic ingestion. In this example, the soil concentration ... risk assessment issues. The WHO has. CHAPTER 4 RISK ASSESSMENT 121.
Contents
Unit 2 Disposition of Toxicants | 129 |
Unit 3 NonOrganDirected Toxicity | 327 |
Unit 4 Target Organ Toxicity | 453 |
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absorption acid activity addition agents alterations animals appears aromatic assays associated binding blood cancer carcinogens catalyzed cause cells cellular changes chemical chromosome compartment compounds concentration conjugation contain cytochrome P450 damage decrease detected determined developmental differences dose drugs effects elimination enzymes et al evaluation example excretion exposure expression factors Figure formation function gene genetic glucuronide glutathione growth half-life Health human important increased individuals induced inhibition inhibitors initial interactions involved known lead levels liver lung major mechanisms membrane metabolism metabolites mice molecules mouse mutagenic mutations normal observed occur organ oxidase oxidative oxygen pathway Pharmacol placenta plasma potential produce proteins rats reactions reactive receptor reduction relatively repair response risk risk assessment role shown signaling species structure studies substrates Table tion tissue toxicity Toxicol toxicology transport tumor vitro whereas xenobiotics