The Pharmacological Basis of TherapeuticsDrugs acting on the central nervous system; Local anesthetics; Drugs acting at synaptic and neuroeffector junctional sites; Autacoids; Cardiovascular drugs; Water, salts, and ions; Drugs affecting renal function and electrolyte metabolism; Drugs affecting uterine motility; Gases and vapors; Heavy metals and heavy-metal antagonists; Locally acting drugs; Chemotherapy of parasitic diseases; Chemotherapy of microbial diseases; Chemotherapy of neoplastic diseases; Drugs acting on the blood and the blood-forming organs; Hormones and hormone antagonists; The vitamins. |
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Page 102
... hypnotic agents are general depressants . They depress a wide range of cellular functions in many vital organ systems . A rational approach to the therapeutic use of hypnotic compounds , and to the treatment of patients poisoned by such ...
... hypnotic agents are general depressants . They depress a wide range of cellular functions in many vital organ systems . A rational approach to the therapeutic use of hypnotic compounds , and to the treatment of patients poisoned by such ...
Page 118
... hypnotic doses will induce sleep that lasts for 6 or 7 hours without interruption . This does not mean that the soporific action of the drug continues unabated for 6 or 7 hours , or that the duration of soporific action is the same for ...
... hypnotic doses will induce sleep that lasts for 6 or 7 hours without interruption . This does not mean that the soporific action of the drug continues unabated for 6 or 7 hours , or that the duration of soporific action is the same for ...
Page 124
... hypnotic use than are needed . Some were developed for other purposes , and their hypnotic use is not the culmination of a search for new hypnotic drugs . These drugs are mostly described elsewhere in this text ( see especially Chapter ...
... hypnotic use than are needed . Some were developed for other purposes , and their hypnotic use is not the culmination of a search for new hypnotic drugs . These drugs are mostly described elsewhere in this text ( see especially Chapter ...
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absorption acid action activity acute addition administration agents alcohol amount analgesic anesthesia anesthetic animals appear associated barbiturates block blood body brain cardiac cause cells central changes Chapter chronic clinical compounds concentration containing convulsions decrease dependence depression develop direct discussed dosage doses drug effects employed enzyme epinephrine et al excreted factors fibers function given heart important increase individual inhibition initial injection intravenous involved less mechanism medication membrane metabolism morphine muscle narcotic nerve nervous normal observed occur opioid oral oxide pain patients period Pharmac pharmacological physical plasma poisoning possible potential preparations present pressure probably produce properties reactions receptors reduced relatively release reported respiratory response result salicylate seizures selective severe side similar solution spinal stimulation structure studies subjects symptoms Ther therapeutic therapy tion tissues tolerance toxic treatment usually various withdrawal