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Mechanisms of HCOT secretion from pancreas and liver An experimental
Head injuries admitted to hospital Epidemiology risk factors
1987 NUMBER 1112
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Acta Physiol Scand active Aker Hospital Al-Awqati alcoholic amiloride aortic Aronson arterial pH ATPase BAIBF basolateral cell membrane bicarbonate bile acid bile duct cells bile flow bile formation biliary bilirubin Biol biopsies blood Buanes bumetanide canalicular carbonic anhydrase cardiac index celiac disease cell membrane Clin Invest clinical control group DCCD diagnosis of idiopathic disease ductular effect of amiloride electrogenic electrolyte endotoxins enzyme epithelia erythritol etiology experimental gastric gastrointestinal investigation Grotmol Haptoglobin HCO^-secretion HCO3 Hemolysis hepatic hepatocytes idiopathic idiopathic polyneuropathy infections inhibited inhibitors jaundice kidney laboratory Mathisen mechanisms Na,K Na,K-ATPase Na+/H+ Na+/H+-exchanger Na+/H+-ion exchanger neurological neuropathy Norway nursing Oslo City Hospitals pancreas and liver pancreatic HCO3-secretion pancreatic secretion pathy polyneuro polyneuropathy postoperative jaundice preoperatively protein proton pump proximal Psychiatry Rceder reabsorption reduced renal role Scharschmidt secretin secretin-dependent Sejersted sodium statistical studies surgery syndrome tion UDCA Ulleval Hospital University of Oslo vesicles Vilming