Cerebrovascular Diseases: Fifteenth Research (Princeton) ConferenceWilliam J. Powers, Marcus E. Raichle |
Contents
Human Performance in the Severe Hypoxia | 1 |
THE BIOCHEMICAL LESION | 11 |
Blockade of Excitatory Neurotransmission Provides Partial | 27 |
Copyright | |
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acidosis activity adenosine amino acid amphetamine animals barrel-field Blood Flow Metab Brain Res calcium capillary carotid artery carotid endarterectomy cerebral blood flow cerebral cortex cerebral ischemia cerebrovascular changes clinical clot concentration coronary cortex cortical damage decrease Department of Neurology diameter disease edema effect excitatory amino acids extracellular focal forebrain function gerbil glucose glucose utilization glutamate hemorrhage hippocampus Hossmann hyperglycemic hypoglycemia hypoxia ICBF increase infarction injection injury ipsilateral lactate lesion M.D. Department measurements membrane metabolism middle cerebral artery Neurology neurons NMDA normal occlusion oxygen patients penumbra perfusion Physiol physiological pial plasma Plum postischemic protein synthesis Pulsinelli Raichle and William ratio Raven Press receptors recirculation recovery region reperfusion rtPA seizures selectively vulnerable Siesjö somatosensory stenosis stimulation streptokinase stroke surgery synaptic therapy thrombolysis thrombus tissue transient ischemic attacks uptake vascular vasodilation vessel vivo York