Magnetic Fields, Vagal Inhibition, and Acetylcholinesterase Activity |
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Magnetic Fields, Vagal Inhibition, and Acetylcholinesterase Activity Wei Young,John W. Gofman No preview available - 1965 |
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15,000-oersted magnetic field 30 minutes 830 oersteds/cm acetylcholine acetylcholinesterase inhibitors ACh hydrolysis rate AChE activity activation of AChE alkali metal ion average gradient bathing fluid medium bathing medium carboxydismutase Cardiac arrhythmia cardiac contractility cardiac rhythm cesium ions contractility and cardiac control rate D-ACh determine Deuterated different vagal heart duration of vagal Effect of magnetic field cessation field was applied h after field hours after cessation hydrolysis rate induced increase in hydrolysis increased ACh hydrolysis INHIBITION CONTROL 40 ions by cesium J. W. Gofman Lawrence Radiation Laboratory liter/sec Magnetic Field Effects Magnetic field oersteds magnetic-field effects marked decrease minutes after cessation minutes after initiation molar moles moles/liter myoneural junction observed effects oersteds cm-1 partial replacement PERIOD OF INHIBITION physostigmine post-field potassium ions rate of ACh replacement of potassium Smith and Cook solid trypsin standard error substrate transducer vagal heart preparation vagal heart system vagal inhibition period Velocity of hydrolysis voltmeter