Textbook of Respiratory Medicine, Volume 2John Frederic Murray, Jay A. Nadel |
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Page 1414
... Normal Afterload . Normal right ventricular after- load is difficult to define and impossible to measure . It represents the impedance , from within and with- out , to the wall motion necessary for the generation of pulsatile blood flow ...
... Normal Afterload . Normal right ventricular after- load is difficult to define and impossible to measure . It represents the impedance , from within and with- out , to the wall motion necessary for the generation of pulsatile blood flow ...
Page 1955
... normal FEV1 / FVC . The forced expiratory flow - volume curve is often truncated near RV when expiratory muscle weakness is severe . 124 The MVV is almost always below normal . 122 , 123 , 125 , 128 , 130-132 , 135 , 140 The TLC is ...
... normal FEV1 / FVC . The forced expiratory flow - volume curve is often truncated near RV when expiratory muscle weakness is severe . 124 The MVV is almost always below normal . 122 , 123 , 125 , 128 , 130-132 , 135 , 140 The TLC is ...
Page 2008
... Normal values for pulmonary vascular resistance ( PVR ) range from 150-250 dynes / second / cm - 5 . In the normal lung , cardiac output can in- crease two- to threefold with no increase in pul- monary artery pressure , i.e. , PVR ...
... Normal values for pulmonary vascular resistance ( PVR ) range from 150-250 dynes / second / cm - 5 . In the normal lung , cardiac output can in- crease two- to threefold with no increase in pul- monary artery pressure , i.e. , PVR ...
Contents
Section J NEOPLASMS OF THE LUNGS | 1169 |
Section A ANATOMY AND DEVELOPMENT OF THE RESPIRATORY | 1236 |
Miscellaneous Malignant Tumors | 1251 |
Copyright | |
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Other editions - View all
Textbook of Respiratory Medicine John F. Murray,Robert J. Mason,Homer A. Boushey No preview available - 2000 |
Common terms and phrases
abnormalities acute airway alveolar proteinosis antibodies antigen Appl arteriovenous asbestos associated asthma biopsy blood flow bronchial bronchogenic carcinoma capillary cardiac output cause cell carcinoma cell lung cancer chemotherapy chest radiograph chronic Clin clinical cor pulmonale cyclophosphamide deep venous thrombosis develop diagnosis dioxide disorders drug dust dyspnea effects embolism Engl eosinophilic exposure factors fibrosing alveolitis fistulas fluid granuloma granulomatosis heart hemoptysis heparin high altitude histologic hypoxia increased infiltrates inhalation interstitial involvement lavage lesions lung cancer lung disease lung injury lymph lymphocytes malignant mechanisms metastases microvascular monary normal obstruction occur oxygen patients perfusion permeability Physiol plasma pleural pneumonitis pollutants primary pulmonary hypertension protein pulmo pulmonary alveolar proteinosis pulmonary artery pulmonary edema pulmonary embolism pulmonary fibrosis pulmonary function pulmonary vascular reported resection Respir respiratory response rheumatoid risk roentgenogram sarcoidosis scan serum silicosis small cell studies survival symptoms syndrome systemic Thorac tion tissue toxicity treatment tumor usually vasculitis vessels