Intensive Care and Emergency Medicine: 4th International SymposiumJ. L. Vincent The significance of a change in Pa02 occuring in a patient with cardiovascular instability cannot be ascertained unless the values for PV0 and CV02 are known. A fall in Pa02 could 2 reflect worsening of pulmonary function or deterioration of cardiac output (with resultant increase in systemic oxygen extraction). PEEP/CPAP would be an appropriate therapy in the former case but frankly deleterious in the latter if applied prior to cardiovascular stabiliza tion. References 1. Douglas ME, Downs JB, Dannemiller FJ et al (1976) Change in pulmonary venous admixture with varying inspired oxygen. Anesthesia and Analgesia 55:688-695 2. Kelman GR, Nunn JF, Prys-Roberts C et al (1967) The influence of cardiac output on arterial oxygena tion. A theoretical study. Br J Anaesth 39:450-458 3. Kirby RR (1986) Respiratory vs cardiovascular dysfunction - How can we differentiate? ASA Refresher Course Lectures, 102 4. Nunn JF (1977) Applied respiratory physiology (2nd ed). Butterworth et Co 5. Van Aken H, Lawin P (1981) Der EinfluB des Herzminutenvolumens auf die arterielle Oxygenation. |
Contents
Role of Eicosanoids in the Development of ARDS | 14 |
Dr Istituto G Anesthesiology Via F Storza | 35 |
Full VS Partial Ventilatory Support | 36 |
Barotrauma K Hillman | 52 |
The Role of Central and Peripheral a and ßAdrenoceptors | 67 |
Contractile Function of the Ischemic Myocardium | 79 |
Echocardiography in the Coronary Care Unit M L Simoons | 108 |
Utilization of Energy Substrate in Critically Ill Patients | 123 |
Colloids and Crystalloids E C Rackow | 136 |
Cerebral Oedema in HeadInjured Patients J D Miller | 149 |
Influence of Temperature on Arterial Blood Gases | 156 |
Antidotes in the Treatment of Acute Poisoning R Askenasi | 169 |
Improving the Use of Intensive Care W Knaus | 185 |
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Common terms and phrases
abnormalities acid activity acute myocardial infarction acute respiratory failure adult respiratory distress airway alveolar arachidonic ARDS arterial oxygenation blood flow breathing capillary cardiac output catheter cause cells cellular chest clinical coagulation colloid complement complications controlled coronary critically ill cyclooxygenase decreased dialysis disease dobutamine dose drugs edema effects embolism endotoxemia endotoxin endotoxin shock experimental factors fibrin fibrinolysis FIO2 fluid function gas exchange glucose heart hemodynamic hemorrhage hetastarch histamine hypertension hypotension hypoxemia improvement increase infection infusion inhibition injury inotropic interstitial intravascular intravenous intubation left ventricular leukocytes leukotrienes lidocaine lung measurements mechanical ventilation mediators membrane metabolic metoprolol mmHg monitoring mortality myocardial infarction naloxone normal oxygen PaCO2 PaO2 patients PEEP perfusion physiologic plasma platelet prostaglandins pulmonary artery pulmonary vascular reduced release renal respiratory distress syndrome response resuscitation role shunt ẞ-adrenoceptor studies Surg surgery surgical techniques therapy thromboxane tion tissue trauma treatment vascular permeability vasoconstriction ventilatory ventricular fibrillation Venturi mask