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Normal and Pathological Aging J E Seegmiller
Neuroendocrinology of Stress and Aging
Grafting Genetically Modified Cells to the Brain
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abnormal Acad acid activity addition adult affinity aging altered Alzheimer Alzheimer's disease amyloid antibody appears associated astrocytes bFGF binding Biochem blood brain cDNA cells changes chromosome compared Congo red containing corticosterone culture decrease dementia demonstrated deposits detected determined diffuse plaques disease dopamine Down's syndrome effect enzyme ethanol evidence exon expression factors findings function further gene genetic glucose hippocampus human immunostaining important increase indicate inhibition inhibitor involved L-DOPA labeling lesions levels loss major mechanism method molecular mRNA Nagatsu Nature neuritic neurons Neurosci normal observed occur old rats patients positive possible present Proc produce protease protein recent receptor region regulation reported response role Science secretion sequence showed shown similar specific staining stress structures studies suggest syndrome tissue treatment young