Shock in Low-and High-flow States: Proceedings of a Symposium at Brook Lodge, Augusta, Michigan, U.S.A. June 1, 2, and 3, 1971Bernard K. Forscher, Richard C. Lillehei, Samuel S. Stubbs |
Contents
23DPG What role in septic shock? | 42 |
Does bradykinin play a pathogenetic role | 59 |
Blood flow and metabolism in brain and in adipose tissue dur | 65 |
Copyright | |
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Common terms and phrases
acid activity acute addition agents albumin alterations Amer animals appears arterial arterial pressure associated blood flow blood pressure capillary cardiac index cardiac output cause cell central venous changes circulation clinical concentration damage decrease demonstrated depressant determined developed difference dogs doses early edema effect endotoxin enzyme evidence experimental factors fluid function given glucose hemodynamic hemorrhagic shock hypotension important improved increased indicate infusion initial injection injury intravenous lactate less leukocytes lung lysosomal mean measurements mechanism membrane metabolic methylprednisolone minutes muscle myocardial myocardial infarction normal observed occur organisms oxygen oxygen consumption patients perfusion period peripheral resistance phenoxybenzamine Physiol physiologic plasma platelet present produced pulmonary release resistance respiratory response role samples sepsis septic shock severe showed shown significant steroids studies suggest Surg Surgery Table therapy tissue trauma treated treatment values vascular venous pressure volume