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ACTIVATION OF THE FIRST COMPONENT OF COMPLEMENT Verne
HISTOINCOMPATIBLE BONE MARROW TRANSPLANTS IN HUMANS
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Acad acid activation addition analysis anti antibodies antigen antigen receptor appears associated binding block blood bone c-myc cell surface cells chromosome clones complex containing cytoplasmic cytotoxic demonstrated detected determinants differentiation disease effects encoded enhancer epitopes evidence experiments expression factor Figure function gene genetic human immune immunoglobulin Immunol important increased indicate induced infection inhibit interaction involved isolated leukemia levels lines locus lymph lymphocytes major marrow mature mechanism mediated membrane mice molecular molecules monoclonal mouse murine mutation Natl Nature normal observed occur patients peptide phenotype possible present Proc production proliferation protein rearrangement recently recognition recognized recombinant region regulation reported response restricted rheumatoid role secretion segments sequence showed shown signal similar specific stimulation strains structure studies suggest suppressor T-cell thymic thymocytes thymus transcription transgenic translocations transplantation vaccinia virus vitro