Semblance Hypothesis of Memory: 3rd edition
The semblance hypothesis explains how associative memory can result from the multidimensional semblance of activity from a specific set of presynaptic terminals at different orders of neurons. The hypothesized basic units are then examined for their feasibility to explain various physiological and pathological conditions of the nervous system, ranging from physiological time-scales of memory retrieval to plausible explanation for long term potentiation (LTP).
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action potential Alzheimer’s disease amygdala associative learning axon axon hillock brain CA1 neuron computational convergence cortex cortical cue stimulus dendritic spines different orders dopamine EPSP-spread evoke explained extracellular fMRI functional LINKs formed functionally LINKed postsynapses granule neurons hallucinations higher orders hippocampus increase induce semblance islet of LINKed islets of functional large number lead learned item LINK formation locations long-term memory memory retrieval memory storage mirror neuron network semblance neural neurogenesis neuronal activity Neurosci novel cue ofthe orders of neurons oscillations oxygen partial neuronal network physiological time-scales possible present hypothesis presynaptic terminal protein re-activation receptor repetition of learning retrieval of memories semblance for memory semblance formed semblance hypothesis semblances occurring sensory inputs sensory receptors sensory stimulus set of postsynapses specific cue specific memory spread of EPSP studies subset of postsynapses summated synapses synaptic and network synaptic plasticity synaptic semblance synthetic premises take place temporal unrelated learning virtual sensation