Robbins and Cotran Pathologic Basis of Disease, Volume 1First Prize (Basic and Clinical Sciences), 2005 BMA Awards Through six editions, Robbins Pathologic Basis of Disease has been the leading textbook in its field. No other resource has been able to match its outstanding coverage of pathophysiology and disease. Now renamed Robbins and Cotran Pathologic Basis of Disease, the new, 7th Edition of this classic text delivers a host of improvements that make it an even clearer choice for students and practitioners! The smart way to study! Elsevier titles STUDENT CONSULT will help you master difficult concepts and study more efficiently in print and online! Perform rapid searches. Integrate bonus content from other disciplines. Download text to your handheld device. And a lot more. Each STUDENT CONSULT title comes with full text online, a unique image library, case studies, USMLE style questions, and online note-taking to enhance your learning experience. Your purchase of this book entitles you to access www.studentconsult.com at no extra charge. This innovative web site offers you...
And now, Pathologic Basis of Disease is also ONLINE! For the life of the edition, readers can access...
It all adds up to a phenomenal New Edition that will continue to assist a wide variety of readers in mastering all of today's most essential pathology knowledge. |
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User Review - bookshoper - Overstock.comI needed this book for school and the best price I found was here. The shipping cost was the lowest of all also.There is no entertainment value to this book as it is a med school book but the priceshipping and quality could not be beat. The book was in perfect condition. Read full review
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abnormal accumulation acid activation acute addition adhesion affected agents antibodies antigens apoptosis appear associated bacteria become binding blood body bone called cancer cause cells cellular changes Chapter chromosome chronic clinical collagen common complement complex contain cytokines cytoplasm damage death defects deficiency deposition described differentiation discussed disease disorders endothelial cells enzymes example expression extracellular FIGURE formation function gene genetic glucose growth factors healing host human immune important increased individuals induced infection inflammation inflammatory inherited initial injury involved known leading leukocytes levels liver loss lymphocytes lysosomal macrophages major mechanisms mediators membrane molecular molecules muscle mutations myocardial infarction necrosis neutrophils normal occur organs pathway patients plasma platelet present proliferation proteins reactions receptors reduced regulation release repair response result role severe signaling skin specific stem cells stimulate structure surface syndrome synthesis tion tissue tumor usually vascular vessels virus wound